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HAL Antikörper (C-Term)

Dieses Kaninchen Polyklonal-Antikörper erkennt spezifisch HAL in WB und IHC. Er zeigt eine Reaktivität gegenüber Human, Maus und Ratte.
Produktnummer ABIN629752

Kurzübersicht für HAL Antikörper (C-Term) (ABIN629752)

Target

Alle HAL Antikörper anzeigen
HAL (Histidine Ammonia-Lyase (HAL))

Reaktivität

  • 19
  • 8
  • 7
  • 4
  • 3
  • 3
  • 2
  • 2
  • 2
  • 1
  • 1
Human, Maus, Ratte

Wirt

  • 16
  • 3
Kaninchen

Klonalität

  • 18
  • 1
Polyklonal

Konjugat

  • 15
  • 2
  • 1
  • 1
Dieser HAL Antikörper ist unkonjugiert

Applikation

  • 14
  • 8
  • 6
  • 3
  • 1
  • 1
Western Blotting (WB), Immunohistochemistry (IHC)
  • Bindungsspezifität

    • 5
    • 4
    • 2
    • 2
    • 1
    • 1
    • 1
    C-Term

    Spezifität

    HAL antibody was raised against the C terminal of HAL

    Aufreinigung

    Purified

    Immunogen

    HAL antibody was raised using the C terminal of HAL corresponding to a region with amino acids EAAHRLLLEQKVWEVAAPYIEKYRMEHIPESRPLSPTAFSLQFLHKKSTK
  • Applikationshinweise

    WB: 1.25 µg/mL, IHC: 4-8 µg/mL
    Optimal conditions should be determined by the investigator.

    Kommentare

    HAL Blocking Peptide, (ABIN937635), is also available for use as a blocking control in assays to test for specificity of this HAL antibody

    Beschränkungen

    Nur für Forschungszwecke einsetzbar
  • Format

    Lyophilized

    Rekonstitution

    Lyophilized powder. Add distilled water for a 1 mg/mL concentration of HAL antibody in PBS

    Konzentration

    Lot specific

    Buffer

    PBS

    Handhabung

    Avoid repeated freeze/thaw cycles.
    Dilute only prior to immediate use.

    Lagerung

    4 °C/-20 °C

    Informationen zur Lagerung

    Store at 2-8 °C for short periods. For longer periods of storage, store at -20 °C.
  • Target

    HAL (Histidine Ammonia-Lyase (HAL))

    Andere Bezeichnung

    HAL

    Hintergrund

    HAL is a cytosolic enzyme catalyzing the first reaction in histidine catabolism, the nonoxidative deamination of L-histidine to trans-urocanic acid. HAL defects cause histidinemia which is characterized by increased histidine and histamine and decreased urocanic acid in body fluidsHistidine ammonia-lyase is a cytosolic enzyme catalyzing the first reaction in histidine catabolism, the nonoxidative deamination of L-histidine to trans-urocanic acid. Histidine ammonia-lyase defects cause histidinemia which is characterized by increased histidine and histamine and decreased urocanic acid in body fluids.

    Molekulargewicht

    72 kDa (MW of target protein)
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