GABA Antikörper
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- Target Alle GABA Antikörper anzeigen
- GABA (gamma-Aminobutyric Acid (GABA))
- Reaktivität
- Bitte anfragen
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Wirt
- Kaninchen
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Klonalität
- Polyklonal
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Konjugat
- Dieser GABA Antikörper ist unkonjugiert
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Applikation
- ELISA, Immunohistochemistry (Frozen Sections) (IHC (fro)), Immunohistochemistry (Paraffin-embedded Sections) (IHC (p)), Immunofluorescence (Paraffin-embedded Sections) (IF (p))
- Kreuzreaktivität
- Ratte
- Kreuzreaktivität (Details)
- GABA
- Aufreinigung
- Purified by Protein A.
- Immunogen
- KLH conjugated GABA
- Isotyp
- IgG
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- Applikationshinweise
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IHC-P 1:100-500
IF(IHC-P) 1:50-200 - Beschränkungen
- Nur für Forschungszwecke einsetzbar
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- Format
- Liquid
- Konzentration
- 1 μg/μL
- Buffer
- 0.01M TBS( pH 7.4) with 1 % BSA, 0.02 % Proclin300 and 50 % Glycerol.
- Konservierungsmittel
- Sodium azide
- Vorsichtsmaßnahmen
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.
- Lagerung
- 4 °C,-20 °C
- Informationen zur Lagerung
- Shipped at 4°C. Store at -20°C for one year. Avoid repeated freeze/thaw cycles.
- Haltbarkeit
- 12 months
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Niche astrocytes promote the survival, proliferation and neuronal differentiation of co-transplanted neural stem cells following ischemic stroke in rats." in: Experimental and therapeutic medicine, Vol. 13, Issue 2, pp. 645-650, (2017) (PubMed).
: "
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Niche astrocytes promote the survival, proliferation and neuronal differentiation of co-transplanted neural stem cells following ischemic stroke in rats." in: Experimental and therapeutic medicine, Vol. 13, Issue 2, pp. 645-650, (2017) (PubMed).
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- Target
- GABA (gamma-Aminobutyric Acid (GABA))
- Andere Bezeichnung
- Gaba (GABA Produkte)
- Substanzklasse
- Amino Acid
- Hintergrund
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Synonyms: gamma-aminobutyric acid, gamma aminobutyric acid, 4 aminobutanoic acid, GABA, Gamma amino butyric acid.
Background: Gamma-aminobutyric acid (GABA) is a major inhibitory neurotransmitter. GABA acts at inhibitory synapses in the brain and spinal cord. Inhibition is provoked by GABA binding resulting in hyperpolarization of the synaptic transmembrane potential of the affected neuron. GABA binding causes ion channels to open allowing either the flow of chloride or potassium ions into or out of the cell.
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