GPR172A antikoerper, MGC79729 antikoerper, GPR172B antikoerper, gpcr41 antikoerper, GPCR42 antikoerper, PAR2 antikoerper, RBFVD antikoerper, RFT1 antikoerper, RFVT1 antikoerper, hRFT1 antikoerper, Gpr172a antikoerper, Gpr172b antikoerper, RGD1560410 antikoerper, rRFT1 antikoerper, POPAR antikoerper, 2010003P03Rik antikoerper, D15Ertd747e antikoerper, mRFT1 antikoerper, solute carrier family 52 member 1 antikoerper, solute carrier family 52 member 2 antikoerper, solute carrier family 52 (riboflavin transporter), member 1 antikoerper, solute carrier family 52 (riboflavin transporter), member 1 L homeolog antikoerper, solute carrier protein 52, member 2 antikoerper, SLC52A1 antikoerper, SLC52A2 antikoerper, slc52a1 antikoerper, slc52a1.L antikoerper, Slc52a2 antikoerper
Hintergrund
Biological redox reactions require electron donors and acceptor. Vitamin B2 is the source for the flavin in flavin adenine dinucleotide (FAD) and flavin mononucleotide (FMN) which are common redox reagents. This gene encodes a member of the riboflavin (vitamin B2) transporter family. Haploinsufficiency of this protein can cause maternal riboflavin deficiency. Multiple alternatively spliced variants, encoding the same protein, have been identified.