Telefon:
+49 (0)241 95 163 153
Fax:
+49 (0)241 95 163 155
E-Mail:
orders@antikoerper-online.de

MAGE-Like 2 Antikörper

MAGEL2 Reaktivität: Human ELISA, IHC Wirt: Kaninchen Polyclonal unconjugated
Produktnummer ABIN7001382
  • Target Alle MAGE-Like 2 (MAGEL2) Antikörper anzeigen
    MAGE-Like 2 (MAGEL2)
    Reaktivität
    • 33
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    Human
    Wirt
    • 33
    Kaninchen
    Klonalität
    • 33
    Polyklonal
    Konjugat
    • 8
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    Dieser MAGE-Like 2 Antikörper ist unkonjugiert
    Applikation
    • 13
    • 13
    • 11
    • 6
    • 6
    • 5
    • 3
    • 1
    ELISA, Immunohistochemistry (IHC)
    Produktmerkmale
    Polyclonal Antibody
    Aufreinigung
    Affinity purification
    Immunogen
    Synthetic peptide of human MAGEL2
    Isotyp
    IgG
    Top Product
    Discover our top product MAGEL2 Primärantikörper
  • Applikationshinweise
    IHC 1:50-1:200
    Beschränkungen
    Nur für Forschungszwecke einsetzbar
  • Format
    Liquid
    Konzentration
    0.8 mg/mL
    Buffer
    PBS with 0.05 % sodium azide and 50 % glycerol, PH7.4
    Konservierungsmittel
    Sodium azide
    Vorsichtsmaßnahmen
    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    Lagerung
    -20 °C
    Informationen zur Lagerung
    Store at -20°C. Avoid freeze / thaw cycles.
  • Target
    MAGE-Like 2 (MAGEL2)
    Andere Bezeichnung
    MAGEL2 (MAGEL2 Produkte)
    Synonyme
    NDNL1 antikoerper, nM15 antikoerper, MAGEL2 antikoerper, Mage-l2 antikoerper, ns7 antikoerper, MAGE family member L2 antikoerper, melanoma antigen, family L, 2 antikoerper, MAGEL2 antikoerper, Magel2 antikoerper
    Hintergrund
    Prader-Willi syndrome (PWS) is caused by the loss of expression of imprinted genes in chromosome 15q11-q13 region. Affected individuals exhibit neonatal hypotonia, developmental delay, and childhood-onset obesity. Necdin (NDN), a gene involved in the terminal differentiation of neurons, localizes to this region of the genome and has been implicated as one of the genes responsible for the etiology of PWS. This gene is structurally similar to NDN, is also localized to the PWS chromosomal region, and is paternally imprinted, suggesting a possible role for it in PWS.
    NCBI Accession
    NP_061939
    UniProt
    Q9UJ55
Sie sind hier:
Kundenservice