SMCR7L Antikörper
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- Target Alle SMCR7L Antikörper anzeigen
- SMCR7L (Smith-Magenis Syndrome Chromosome Region, Candidate 7-Like (SMCR7L))
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Reaktivität
- Human, Maus, Ratte
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Wirt
- Kaninchen
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Klonalität
- Polyklonal
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Konjugat
- Dieser SMCR7L Antikörper ist unkonjugiert
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Applikation
- Western Blotting (WB), ELISA, Immunofluorescence (IF), Immunoprecipitation (IP)
- Aufreinigung
- Immunogen affinity purified
- Reinheit
- ≥95 % as determined by SDS-PAGE
- Immunogen
- Smith-Magenis syndrome chromosome region, candidate 7-like
- Isotyp
- IgG
- Top Product
- Discover our top product SMCR7L Primärantikörper
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- Applikationshinweise
- WB: 1:500-1:5000, IP: 1:500-1:5000, IF: 1:10-1:100
- Beschränkungen
- Nur für Forschungszwecke einsetzbar
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- Format
- Liquid
- Buffer
- PBS with 0.02 % sodium azide and 50 % glycerol pH 7.3,
- Konservierungsmittel
- Sodium azide
- Vorsichtsmaßnahmen
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Lagerung
- -20 °C
- Informationen zur Lagerung
- -20°C for 12 months (Avoid repeated freeze / thaw cycles.)
- Haltbarkeit
- 12 months
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- Target
- SMCR7L (Smith-Magenis Syndrome Chromosome Region, Candidate 7-Like (SMCR7L))
- Andere Bezeichnung
- SMCR7L (SMCR7L Produkte)
- Synonyme
- MGC115280 antikoerper, HSU79252 antikoerper, MID51 antikoerper, MIEF1 antikoerper, dJ1104E15.3 antikoerper, A230016E22 antikoerper, AI452372 antikoerper, im:7156228 antikoerper, smcr7l antikoerper, zgc:162279 antikoerper, mitochondrial elongation factor 1 antikoerper, mitochondrial elongation factor 1 S homeolog antikoerper, MIEF1 antikoerper, mief1.S antikoerper, Mief1 antikoerper, mief1 antikoerper
- Hintergrund
- Synonyms:dJ1104E15.3, FLJ20232, HSU79252, MID51, MIEF1, SMCR7L Background:Human SMCR7L gene encodes, MID51, the mitochondrial dynamic protein of 51 kDa(also called mitochondrial elongation factor 1, MIEF1). MID51 is a single-pass membrane protein anchored to the mitochondrial outer membrane and regulates mitochondrial morphology. Mitochondrial morphology is controlled by two opposing processes: fusion and fission. Elevated MID51 levels induce extensive mitochondrial fusion, whereas depletion of MID51 causes mitochondrial fragmentation. MID51 interacts with and recruits Drp1 to mitochondria, suggesting a critical role of MID51 in regulation of mitochondrial fusion-fission machinery in vertebrates.
- Molekulargewicht
- 49 kDa
- UniProt
- Q9NQG6
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